Cell apoptosis, sometimes called programmed cell death, is a cellular self-destruction method to remove old and damaged cells during development and aging to protect cells from external disturbances and maintain homeostasis. Apoptosis also occurs as a defense mechanism such as in immune reactions or when cells are damaged by disease or noxious agents.

Apoptosis is controlled by many genes and involves two fundamental pathways: the extrinsic pathway, which transmits death signals by the death receptor (DR), and the intrinsic or mitochondrial pathway. The extrinsic apoptotic pathway is activated by the binding of the death ligand to DRs, including FasL, TNF-α, and TRAIL, on the plasma membrane. The DR, adaptor protein (FADD), and associated apoptosis signaling molecule (caspase-8) form the death-inducing signaling complex (DISC), thus leading to the activation of the effector caspase cascade (caspase-3, -6, and -7). The mitochondria-mediated intrinsic apoptosis pathway is regulated by Bcl-2 family proteins, including proapoptotic (Bid, Bax, Bak) and antiapoptotic proteins (Bcl-2, Bcl-xL).

Abnormalities in cell apoptosis can be a significant component of diseases such as cancer, autoimmune lymphoproliferative syndrome, AIDS, ischemia, and neurode-generative diseases. These diseases may benefit from artificially inhibiting or activating apoptosis. A short list of potential methods of anti-apoptotic therapy includes stimulation of the IAP (inhibitors of apoptosis proteins) family of proteins, caspase inhibition, PARP (poly [ADP-ribose] polymerase) inhibition, stimulation of the PKB/Akt (protein kinase B) pathway, and inhibition of Bcl-2 proteins.

Ferroptosis and necroptosis are recently recognized forms of regulated cell death that differs considerably from apoptosis. Misregulated ferroptosis or necroptosis have also been implicated in multiple physiological and pathological processes, including cancer cell death, neurotoxicity, neurodegenerative diseases, etc.

References:
[1] Susan Elmore. Toxicol Pathol. 2007; 35(4): 495–516.
[2] Cao L, et al. J Cell Death. 2016 Dec 29;9:19-29.
[3] Dasgupta A, et al. Int J Mol Sci. 2017 Jan; 18(1): 23.
[4] Xie Y, et al. Cell Death Differ. 2016 Mar;23(3):369-79.


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Clitocine

Clitocine, an adenosine nucleoside analog, is a potent and efficacious readthrough agent. Clitocine can induce the production of p53 protein in cells harboring p53 nonsense-mutated alleles. Clitocine can induce apoptosis in multidrug-resistant human cancer cells by targeting Mcl-1. Anticancer activity[1][2].

  • CAS Number: 105798-74-1
  • MF: C9H13N5O6
  • MW: 287.22900
  • Catalog: Apoptosis
  • Density: 1.82g/cm3
  • Boiling Point: 702.4ºC at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 378.6ºC

Nampt-IN-3

Nampt-IN-3 (Compound 35) simultaneously inhibit nicotinamide phosphoribosyltransferase (NAMPT) and HDAC with IC50s of 31 nM and 55 nM, respectively. Nampt-IN-3 effectively induces cell apoptosis and autophagy and ultimately leads to cell death[1].

  • CAS Number: 2121591-52-2
  • MF: C29H25N7O2
  • MW: 503.55
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

DB1113

DB1113 (Example 24) is a bifunctional compound targeted protein degradation of kinases. DB1113 degrades ABL1, ABL2, BLK, CDK11B, CDK4, CSK, EPHA3, FER, GAK, LIMK1, MAP3K20, MAP4K1, MAP4K2, MAP4K3, MAP4K5, MAPK14, MAPK7, MAPK8, MAPK9, MAPKAPK2, MAPKAPK3, NLK, PDIK1L, PTK2B, RIPK1, RPS6KA1, RPS6KA3, SIK2, SIK3, STK35, TNK2, and ULK1. DB1113 can be used for research of disease or disorder mediated by aberrant kinase activity[1].

  • CAS Number: 2769753-53-7
  • MF: C59H68F3N13O6S
  • MW: 1144.31
  • Catalog: Ferroptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Sodium Oleate

Sodium oleate (Oleic acid sodium) is an abundant monounsaturated fatty acid sodium[1]. Sodium oleate is a Na+/K+ ATPase activator[2].

  • CAS Number: 143-19-1
  • MF: C18H33NaO2
  • MW: 304.443
  • Catalog: Apoptosis
  • Density: 0.9 g/cm3
  • Boiling Point: 359.999ºC at 760 mmHg
  • Melting Point: 232-235 °C(lit.)
  • Flash Point: 270.099ºC

RIP2 kinase inhibitor 2

RIP2 kinase inhibitor 2 is a receptor interacting protein-2 (RIP2) kinase inhibitor extracted from patent WO/2014043437 A1, compound example 9.

  • CAS Number: 1581270-11-2
  • MF: C21H28N4O4S
  • MW: 432.536
  • Catalog: RIP kinase
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 670.0±55.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 359.0±31.5 °C

RIPA-56

RIPA-56 is a highly potent, selective, and metabolically stable inhibitor of receptor-interacting protein 1 (RIP1) with an IC50 of 13 nM.

  • CAS Number: 1956370-21-0
  • MF: C13H19NO2
  • MW: 221.30
  • Catalog: RIP kinase
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Licofelone-d4

Licofelone-d4 (ML-3000-d4) is the deuterium labeled Licofelone. Licofelone (ML-3000) is a dual COX/5-lipoxygenase (5-LOX) inhibitor (IC50=0.21/0.18 μM, respectively) for the treatment of osteoarthritis. Licofelone exerts anti-inflammatory and anti-proliferative effects. Licofelone induces apoptosis, and decreases the production of proinflammatory leukotrienes and prostaglandins[1][2][3].

  • CAS Number: 1189427-04-0
  • MF: C23H18D4ClNO2
  • MW: 383.90
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Tezacitabine

Tezacitabine is a cytostatic and cytotoxic antimetabolite and a nucleoside analogue. Tezacitabine irreversibly inhibits the ribonucleotide reductase and interferes with DNA replication and repair. Tezacitabine effectively induces cells apoptotic. Tezacitabine has the potential for leukemias and solid tumors (carcinomas) treatment[1][2].

  • CAS Number: 130306-02-4
  • MF: C10H12FN3O4
  • MW: 275.234
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: 590.1ºC at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 310.7ºC

BCP-T.A

BCP-T.A, a tunable heterocyclic electrophile, is a potent ferroptosis inducer by binding to GPX4[1].

  • CAS Number: 2786829-70-5
  • MF: C23H19Cl2N3OS
  • MW: 456.39
  • Catalog: Ferroptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Anticancer agent 76

Anticancer agent 76 (Compound CT2-3) is an anticancer agent. Anticancer agent 76 significantly inhibits the proliferation of human NSCLC cells, induces cell cycle arrest, causes ROS generation and induces cell apoptosis[1].

  • CAS Number: 2448091-11-8
  • MF: C32H33NO5S
  • MW: 543.67
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

GSK2593074A

GSK2593074A (GSK’074) is a necroptosis inhibitor with dual targeting ability to both RIP1 and RIP3[1].

  • CAS Number: 1337531-06-2
  • MF: C27H23N5OS
  • MW: 465.57
  • Catalog: RIP kinase
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

5β-Dihydrocortisol

5β-Dihydrocortisol, a metabolite of Cortisol, is a potential mineralocorticoid. 5β-Dihydrocortisol can potentiate glucocorticoid activity in raising the intraocular pressure. 5β-Dihydrocortisol causes breast cancer cell apoptosis[1][2][3][4][5].

  • CAS Number: 1482-50-4
  • MF: C21H32O5
  • MW: 364.47600
  • Catalog: Apoptosis
  • Density: 1.249g/cm3
  • Boiling Point: 544.5ºC at 760mmHg
  • Melting Point: N/A
  • Flash Point: 297.1ºC

(R)-2-AMINO-3-(PROP-2-YN-1-YLTHIO)PROPANOIC ACID

S-Propargylcysteine (SPRC), a structural analog of S-allyl cysteine (SAC), is a slow H2S-releasing compound. S-Propargylcysteine reduces Ca2+ accumulation and inflammatory cytokines, inhibits STAT3, and elevates p53 and Bax. S-Propargylcysteine has anti-inflammatory activity and protects mice against acute pancreatitis. S-Propargylcysteine also has cardioprotective, neuroprotective acitivties[1][2].

  • CAS Number: 3262-64-4
  • MF: C6H9NO2S
  • MW: 159.21
  • Catalog: MDM-2/p53
  • Density: 1.284g/cm3
  • Boiling Point: 294.8ºC at 760 mmHg
  • Melting Point: 176-178 °C
  • Flash Point: 132.1ºC

dl-Citronellol acetate

Citronellyl acetate is a monoterpene product of the secondary metabolism of plants, with antinociceptive activity. Citronellyl acetate exhibits pro-apoptotic activity in human hepatoma cells. Citronellyl acetate shows fungicidal, larvicidal, bactericidal and repelling/insecticidal effects[1].

  • CAS Number: 150-84-5
  • MF: C12H22O2
  • MW: 198.302
  • Catalog: Apoptosis
  • Density: 0.9±0.1 g/cm3
  • Boiling Point: 258.5±19.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 88.2±19.9 °C

1',4"-Sophorolactone 6',6"-diacetate

Lactonic sophorolipid is a natural antimicrobial surfactant for oral hygiene[1]. Lactonic sophorolipid, a potential anticancer agent, induces apoptosis in human HepG2 cells through the caspase-3 pathway[1].

  • CAS Number: 148409-20-5
  • MF: C34H56O14
  • MW: 688.800
  • Catalog: Bacterial
  • Density: 1.2±0.1 g/cm3
  • Boiling Point: 843.8±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 256.9±27.8 °C

WDR5-IN-1

WDR5-IN-1 is a potent and selective WD repeat domain 5 (WDR5) inhibitor, with a Kd of <0.02 nM. WDR5-IN-1 inhibits MLL1 histone methyltransferase (HMT) activity with an IC50 of 2.2 nM. WDR5-IN-1 diminishes MYC recruitment at WDR5-displaced genes and exhibits potent anti-proliferative effects in CHP-134 (neuroblastoma) and Ramos (Burkitt’s lymphoma) lines[1].

  • CAS Number: 2408842-51-1
  • MF: C30H31FN4O3
  • MW: 514.59
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

L-Cystine-34S2

L-Cystine-34S2 is the 34S-labeled L-Cystine. L-Cystine is an amino acid and intracellular thiol, which plays a critical role in the regulation of cellular processes.

  • CAS Number: 113512-08-6
  • MF: C6H12N2O434S2
  • MW: 244.11
  • Catalog: Ferroptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

LACTO-N-FUCOPENTAOSE I

Lacto-N-fucopentaose I (LNFPI) is a human milk oligosaccharide (HMO), possessing antiviral and antibacterial activity. Lacto-N-fucopentaose I can reduce capsid protein VP1 to block virus adsorption, promote CDK2 and reduce cyclin E to recover cell cycle S phase block. Lacto-N-fucopentaose I inhibits ROS production and apoptosis in virus-infected cells. Lacto-N-fucopentaose I can also regulate intestinal microbiota to affect immune system development[1].

  • CAS Number: 7578-25-8
  • MF: C32H55NO25
  • MW: 853.77100
  • Catalog: Bacterial
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Jolkinolide A

Jolkinolide A is a diterpenoid, can be extracted from the roots of Euphorbia fischeriana Steud. Jolkinolide A exhibits anti-tumor activity, by affecting on angiogenesis of tumor tissues. Jolkinolide A significantly inhibits the Akt-STAT3-mTOR signaling pathway and reduces the expression of VEGF in A549 cells[1].

  • CAS Number: 37905-07-0
  • MF: C20H26O3
  • MW: 314.41864
  • Catalog: Apoptosis
  • Density: 1.2±0.1 g/cm3
  • Boiling Point: 479.5±45.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 206.7±23.3 °C

HP661

HP661 is an orally available oxidative phosphorylation (OXPHOS) inhibitor that effectively blocks the activity of mitochondrial complex I and reduces the production of the reactive oxygen species. HP661 can abrogate the growth of high OXPHOS-dependent tumors, conquer high OXPHOS-mediated drug resistance, and can induce cell apoptosis. And HP661 can be used for anti-tumor research[1].

  • CAS Number: 2938220-60-9
  • MF: C31H29F6N7O3
  • MW: 661.60
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

BM-1244

BM-1244 is a potent Bcl-xL/Bcl-2 inhibitor with Kis of 134 and 450 nM for Bcl- xL and Bcl-2, respectively. BM-1244 inhibits senescent fibroblasts (SnCs) with an EC50 of 5 nM. (From patent WO2019033119A1)[1].

  • CAS Number: 1619923-32-8
  • MF: C54H59ClF4N6O8S4
  • MW: 1159.79
  • Catalog: Bcl-2 Family
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

TC ASK 10

TC ASK 10 (Compound 10) is a potent, selective and orally active apoptosis signal-regulating kinase 1 (ASK1) inhibitor with an IC50 of 14 nM. The inhibitory activities of TC ASK 10 towards other representative panel of kinases are less than 50%, except for ASK2 (IC50 of 0.51 μM)[1].

  • CAS Number: 1005775-56-3
  • MF: C21H23Cl2N5O
  • MW: 432.346
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Glyphosate-d2

Glyphosate-d2 is the deuterium labeled Glyphosate. Glyphosate is an herbicidal derivative of the amino acid glycine. Glyphosate targets and blocks a plant metabolic pathway not found in animals, the shikimate pathway, required for the synthesis of aromatic amino acids in plants[1].

  • CAS Number: 2733532-11-9
  • MF: C3H6D2NO5P
  • MW: 171.09
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Coenzyme Q10-d6

Coenzyme Q10-d6 is deuterium labeled Coenzyme Q10. Coenzyme Q10 is an essential cofactor of the electron transport chain and a potent antioxidant agent.

  • CAS Number: 110971-02-3
  • MF: C59H84D6O4
  • MW: 869.38
  • Catalog: Ferroptosis
  • Density: 1.0±0.1 g/cm3
  • Boiling Point: 869.0±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 324.6±34.3 °C

(+)-brazilin

Brazilin is a red dye precursor obtained from the heartwood of several species of tropical hardwoods. Brazilin inhibits the cells proliferation, promotes apoptosis, and induces autophagy through the AMPK/mTOR pathway. Brazilin shows chondroprotective and anti-inflammatory activities[1][2][3].

  • CAS Number: 474-07-7
  • MF: C16H14O5
  • MW: 286.279
  • Catalog: Apoptosis
  • Density: 1.6±0.1 g/cm3
  • Boiling Point: 555.8±50.0 °C at 760 mmHg
  • Melting Point: 156-157ºC
  • Flash Point: 289.9±30.1 °C

Mepazine

Mepazine (Pecazine) is a potent and selective MALT1 inhibitor. Mepazine inhibits GSTMALT1 full length and GSTMALT1 325-760 with IC50s of 0.83 and 0.42 μM, respectively. Mepazine affects viability of ABC-DLBCL cells by enhancing apoptosis[1].

  • CAS Number: 60-89-9
  • MF: C19H22N2S
  • MW: 310.45600
  • Catalog: Apoptosis
  • Density: 1.159 g/cm3
  • Boiling Point: 444.4ºC at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 222.6ºC

YH 239-EE

YH239-EE, ethyl ester of the free carboxylic acid compound YH239, is a potent p53-MDM2 antagonizing and apoptosis-inducing agentIC50 value:Target: MDM2/p53YH239-EE inhibits the growth of OCI-AML-3 cells with wild type p53 by inhibiting the p53-MDM2 interaction. YH239-EE induces cell cycle arrest and causes potent cell apoptosis via activation of p53 and downstream targets in four AML cells (OCI-AML-3 and MOLM-13 with wt p53, NB4 with p53 mutation, and HL60 with p53 deletion).

  • CAS Number: 1364488-67-4
  • MF: C25H27Cl2N3O4
  • MW: 504.406
  • Catalog: MDM-2/p53
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 763.3±60.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 415.4±32.9 °C

β-Eleostearic acid

α-Eleostearic acid (cis-Eleostearic acid), a conjugated linolenic acid, is an apoptosis inducer. α-Eleostearic acid is also a ferroptosis inducer. α-Eleostearic acid exhibits antioxidant and antitumor activity[1][2][3].

  • CAS Number: 506-23-0
  • MF: C18H30O2
  • MW: 278.430
  • Catalog: Apoptosis
  • Density: 0.9±0.1 g/cm3
  • Boiling Point: 390.6±11.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 287.4±14.4 °C

Mitoguazone

Mitoguazone (Methylglyoxal-bis(guanylhydrazone)) is a synthetic polycarbonyl derivative with potent antineoplastic activity. Mitoguazone is a brain-penetrant and competitive S-adenosyl-methionine decarboxylase (SAMDC) inhibitor that disrupts polyamine biosynthesis. Mitoguazone induces cell apoptosis. Mitoguazone inhibits HIV DNA integration into the cellular DNA in both monocytes and macrophages. Mitoguazone has the potential for acute leukemia, Hodgkin's and non-Hodgkin's lymphoma treatment[1][2][3][4].

  • CAS Number: 459-86-9
  • MF: C5H12N8
  • MW: 184.20200
  • Catalog: HIV
  • Density: 1.55g/cm3
  • Boiling Point: 436.6ºC at 760mmHg
  • Melting Point: N/A
  • Flash Point: 217.8ºC

W-7 hydrochloride

W-7 hydrochloride is a selective calmodulin antagonist. W-7 hydrochloride inhibits the Ca2+-calmodulin-dependent phosphodiesterase and myosin light chain kinase with IC50 values of 28 μM and 51 µM, respectively[1][2]. W-7 hydrochloride induces apoptosis and has antitumor activity[3].

  • CAS Number: 61714-27-0
  • MF: C16H22Cl2N2O2S
  • MW: 377.329
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: 518.8ºC at 760 mmHg
  • Melting Point: 220-222 °C
  • Flash Point: 267.6ºC