Cell apoptosis, sometimes called programmed cell death, is a cellular self-destruction method to remove old and damaged cells during development and aging to protect cells from external disturbances and maintain homeostasis. Apoptosis also occurs as a defense mechanism such as in immune reactions or when cells are damaged by disease or noxious agents.

Apoptosis is controlled by many genes and involves two fundamental pathways: the extrinsic pathway, which transmits death signals by the death receptor (DR), and the intrinsic or mitochondrial pathway. The extrinsic apoptotic pathway is activated by the binding of the death ligand to DRs, including FasL, TNF-α, and TRAIL, on the plasma membrane. The DR, adaptor protein (FADD), and associated apoptosis signaling molecule (caspase-8) form the death-inducing signaling complex (DISC), thus leading to the activation of the effector caspase cascade (caspase-3, -6, and -7). The mitochondria-mediated intrinsic apoptosis pathway is regulated by Bcl-2 family proteins, including proapoptotic (Bid, Bax, Bak) and antiapoptotic proteins (Bcl-2, Bcl-xL).

Abnormalities in cell apoptosis can be a significant component of diseases such as cancer, autoimmune lymphoproliferative syndrome, AIDS, ischemia, and neurode-generative diseases. These diseases may benefit from artificially inhibiting or activating apoptosis. A short list of potential methods of anti-apoptotic therapy includes stimulation of the IAP (inhibitors of apoptosis proteins) family of proteins, caspase inhibition, PARP (poly [ADP-ribose] polymerase) inhibition, stimulation of the PKB/Akt (protein kinase B) pathway, and inhibition of Bcl-2 proteins.

Ferroptosis and necroptosis are recently recognized forms of regulated cell death that differs considerably from apoptosis. Misregulated ferroptosis or necroptosis have also been implicated in multiple physiological and pathological processes, including cancer cell death, neurotoxicity, neurodegenerative diseases, etc.

References:
[1] Susan Elmore. Toxicol Pathol. 2007; 35(4): 495–516.
[2] Cao L, et al. J Cell Death. 2016 Dec 29;9:19-29.
[3] Dasgupta A, et al. Int J Mol Sci. 2017 Jan; 18(1): 23.
[4] Xie Y, et al. Cell Death Differ. 2016 Mar;23(3):369-79.


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Venetoclax (ABT-199)

Venetoclax (GDC-0199; ABT-199) is a highly potent, selective and orally bioavailable Bcl-2 inhibitor with a Ki of less than 0.01 nM.

  • CAS Number: 1257044-40-8
  • MF: C45H50ClN7O7S
  • MW: 868.439
  • Catalog: Bcl-2 Family
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Beta-Elemonic acid

β-Elemonic acid is a triterpene isolated from Boswellia papyrifera. β-Elemonic acid induces cell apoptosis, reactive oxygen species (ROS) and COX-2 expression and inhibits prolyl endopeptidase. β-Elemonic acid exhibits anticancer and anti-inflammatory effects[1][2].

  • CAS Number: 28282-25-9
  • MF: C30H46O3
  • MW: 454.684
  • Catalog: Apoptosis
  • Density: 1.07
  • Boiling Point: 565.2±50.0 °C at 760 mmHg
  • Melting Point: 216-219 ºC
  • Flash Point: 309.6±26.6 °C

GEM144

GEM144 is a potent and orally active DNA polymerase α (POLA1) and HDAC 11 dual inhibitor. GEM144 induces acetylation of p53, activation of p21, G1/S cell cycle arrest, and apoptosis. GEM144 has significant antitumor activity in human orthotopic malignant pleural mesothelioma xenografts[1].

  • CAS Number: 2487526-28-1
  • MF: C28H31NO5
  • MW: 461.55
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

c16 ceramide

C16-Ceramide is a natural small molecule activating p53 through the direct and selective binding[1].

  • CAS Number: 24696-26-2
  • MF: C34H67NO3
  • MW: 537.90100
  • Catalog: Apoptosis
  • Density: 0.919g/cm3
  • Boiling Point: 675.396ºC at 760 mmHg
  • Melting Point: 94-95ºC
  • Flash Point: 362.267ºC

(6R)-FR054

(6R)-FR054 is an active isomer of FR054. FR054 is an inhibitor of the HBP enzyme PGM3, with a remarkable anti-breast cancer effect. FR054induces endoplasmic reticulum (ER) stress and a ROS-dependent apoptosis[1].

  • CAS Number: 10378-06-0
  • MF: C14H19NO8
  • MW: 329.30300
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

LQZ-7I

LQZ-7I is a survivin-targeting inhibitor. LQZ-7I inhibits survivin dimerization. LQZ-7I orally effectively inhibits xenograft tumor growth and induces survivin loss in tumors[1].

  • CAS Number: 195822-23-2
  • MF: C20H14F2N4
  • MW: 348.35
  • Catalog: Survivin
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Anticancer agent 66

Anticancer agent 66 (Compound 13e) is an anti-cancer agent. Anticancer agent 66 induces apoptosis and increases sub-G1 cell population in MCF-7 cells. Anticancer agent 66 is a ciprofloxacin analog[1].

  • CAS Number: 2691895-64-2
  • MF: C26H23Cl2FN6O2S2
  • MW: 605.53
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Lidocaine HCl

Lidocaine hydrochloride hydrate (Lignocaine hydrochloride hydrate) inhibits sodium channels involving complex voltage and using dependence. Lidocaine hydrochloride hydrate decreases growth, migration and invasion of gastric carcinoma cells via up-regulating miR-145 expression and further inactivation of MEK/ERK and NF-κB signaling pathways. Lidocaine hydrochloride hydrate is an amide derivative and has potential for the research of ventricular arrhythmia[1][2].

  • CAS Number: 6108-05-0
  • MF: C14H25ClN2O2
  • MW: 288.814
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: 350.8ºC at 760 mmHg
  • Melting Point: 68.5ºC
  • Flash Point: 166ºC

2-(3-(2,3-Dichlorophenoxy)propylamino)ethanol hydrochloride

2,3-DCPE induces apoptosis and downregulates Bcl-XL expression. 2,3-DCPE has anti-tumor activity and can be used in cancer research[1].

  • CAS Number: 418788-90-6
  • MF: C11H15Cl2NO2
  • MW: 264.15
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: 402.9ºC at 760mmHg
  • Melting Point: N/A
  • Flash Point: 197.5ºC

δ-secretase inhibitor 11

δ-Secretase inhibitor 11 (compound 11) is an orally active, potent, BBB-penetrated, non-toxic, selective and specific δ-secretase inhibitor, with an IC50 of 0.7 μM. δ-Secretase inhibitor 11 interacts with both the active site and allosteric site of δ-secretase. δ-Secretase inhibitor 11 attenuates tau and APP (amyloid precursor protein) cleavage. δ-Secretase inhibitor 11 ameliorates synaptic dysfunction and cognitive impairments in tau P301S and 5XFAD transgenic mouse models. δ-Secretase inhibitor 11 can be used for Alzheimer's disease research[1].

  • CAS Number: 842964-18-5
  • MF: C10H12N4O2
  • MW: 220.22800
  • Catalog: Caspase
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Cardanol (C15:1)

Cardanol monoene (Cardanol C15:1) is a phenolic compound which can be found in cashew nut shell liquid. Cardanol monoene can induce mitochondria-associated apoptosis in human melanoma cells[1].

  • CAS Number: 501-26-8
  • MF: C21H34O
  • MW: 302.49400
  • Catalog: Apoptosis
  • Density: 0.919g/cm3
  • Boiling Point: 435.4ºC at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 196.6ºC

DB1976 hydrochloride

DB1976 hydrochloride is a selenophene analog of DB270 and a potent and cell-permeable fully efficacious transcription factor PU.1 inhibitor. DB1976 hydrochloride potently inhibits PU.1 binding (IC50 of 10 nM) and strongly inhibits the PU.1/DNA complex (with high DB1976-λB affinity, KD of 12 nM) in vitro. DB1976 hydrochloride has apoptosis-inducing effect[1][2][3].

  • CAS Number: 2369663-93-2
  • MF: C20H18Cl2N8Se
  • MW: 520.28
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

GSK2982772

GSK2982772 is a potent and ATP competitive RIP1 inhibitor with an IC50 of 16 nM.

  • CAS Number: 1622848-92-3
  • MF: C20H19N5O3
  • MW: 377.397
  • Catalog: RIP kinase
  • Density: 1.4±0.1 g/cm3
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

PI3Kδ/γ-IN-3

PI3Kδ/γ-IN-3 (Compound 58) is a potent and orally active PI3Kδ and PI3Kγ dual inhibitor with IC50s of 1 nM and 16 nM, respectively. PI3Kδ/γ-IN-3 induces tumor cell apoptosis and can be used for B-cell malignancies research[1].

  • CAS Number: 2730151-31-0
  • MF: C23H20ClN9O
  • MW: 473.92
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

c-Myc inhibitor 9

c-Myc inhibitor 9 (compound 332) is a c-Myc inhibitor with an logEC50 of ≥6. c-Myc inhibitor 9 inhibits tumor growth in nude mouse models. c-Myc inhibitor 9 can be used for cancer research[1].

  • CAS Number: 2799717-96-5
  • MF: C27H31N5OS
  • MW: 473.63
  • Catalog: c-Myc
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

(+)-Nortrachelogenin

(+)-Nortrachelogenin (Wikstromol), a pharmacologically ligand from from wikstroemia indica, possesses antileukemic activity[1].

  • CAS Number: 61521-74-2
  • MF: C20H22O7
  • MW: 374.384
  • Catalog: Apoptosis
  • Density: 1.4±0.1 g/cm3
  • Boiling Point: 609.2±50.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 217.3±23.6 °C

(2E)-3-(2-hydroxyphenyl)prop-2-enal

2-Hydroxycinnamaldehyde is a phenylpropanoid that can be isolated from the bark of Cinnamomum cassia. 2-Hydroxycinnamaldehyde inhibits Wnt/β-catenin, STAT3 signaling. 2-Hydroxycinnamaldehyde induces cell apoptosis 2-Hydroxycinnamaldehyde has antitumor and anti-inflammation activities[1][2][3].

  • CAS Number: 3541-42-2
  • MF: C9H8O2
  • MW: 148.16
  • Catalog: Apoptosis
  • Density: 1.2±0.1 g/cm3
  • Boiling Point: 311.3±17.0 °C at 760 mmHg
  • Melting Point: 126ºC
  • Flash Point: 131.8±13.5 °C

PU 02

PU02, a derivative of 6-MP (HY-13677), is a negative allosteric modulator (NAM) of 5-HT3 receptor, with IC50 values of 0.36 and 0.73 μM in HEK293 cells transfected with human 5-HT3A and 5-HT3AB receptors respectively[1][2].

  • CAS Number: 313984-77-9
  • MF: C16H12N4S
  • MW: 292.35800
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

BCL6 PROTAC 1

BCL6 PROTAC 1 is a selective B-cell lymphoma 6 (BCL6) PROTAC. BCL6 PROTAC 1 inhibits BCL6 cell reporter with an IC50 value of 8.8 µM. BCL6 PROTAC 1 significantly degrades BCL6 in diffuse large B-cell lymphoma (DLBCL) cell lines. BCL6 PROTAC 1 can be used in tumor related research[1].

  • CAS Number: 2257479-54-0
  • MF: C45H52ClN9O12
  • MW: 946.40
  • Catalog: Bcl-2 Family
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Rilmenidine

Rilmenidine, an innovative antihypertensive agent, is an orally active, selective I1 imidazoline receptor agonist. Rilmenidine is an alpha 2-adrenoceptor agonist. Rilmenidine induces autophagy. Rilmenidine modulates proliferation and stimulates the proapoptotic protein Bax thus inducing the perturbation of the mitochondrial pathway and apoptosis in human leukemic K562 cells[1][2][3].

  • CAS Number: 54187-04-1
  • MF: C10H16N2O
  • MW: 180.247
  • Catalog: Apoptosis
  • Density: 1.5±0.1 g/cm3
  • Boiling Point: 355.5±9.0 °C at 760 mmHg
  • Melting Point: 106 - 107ºC
  • Flash Point: 168.8±18.7 °C

Terrein

Terrein is a melanogenesis inhibitor[1]. Terrein induces apoptosis in breast cancer cell lines [2]. Terrein is an inhibitor of quorum sensing and c-di-GMP in Pseudomonas aeruginosa[3].

  • CAS Number: 582-46-7
  • MF: C8H10O3
  • MW: 154.16300
  • Catalog: Apoptosis
  • Density: 1.449g/cm3
  • Boiling Point: 327.2ºC at 760mmHg
  • Melting Point: N/A
  • Flash Point: 165.9ºC

CPTH2

CPTH2 is a potent histone acetyltransferase (HAT) inhibitor. CPTH2 selectively inhibits the acetylation of histone H3 by Gcn5. CPTH2 induces apoptosis and decreases the invasiveness of a clear cell renal carcinoma (ccRCC) cell line through the inhibition of acetyltransferase p300 (KAT3B)[1][2].

  • CAS Number: 357649-93-5
  • MF: C14H14ClN3S
  • MW: 291.79900
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

SANGUINARINE

Sanguinarine, a benzophenanthridine alkaloid derived from the root of Sanguinaria Canadensis, can stimulate apoptosis via activating the production of reactive oxygen species (ROS). Sanguinarine-induced apoptosis is associated with the activation of JNK and NF-κB.

  • CAS Number: 2447-54-3
  • MF: C20H14NO4
  • MW: 332.329
  • Catalog: Apoptosis
  • Density: 1.3463 (rough estimate)
  • Boiling Point: 483.53°C (rough estimate)
  • Melting Point: 205-215ºC
  • Flash Point: N/A

EGFR-IN-60

EGFR-IN-60 (Compound 7d) shows obvious inhibition of EGFRWT, EGFRT790M, EGFRL858R and JAK3 with IC50s of 83, 26, 53, and 69 nM, respectively. EGFR-IN-60 potently inhibits the growth of H1975 cells harboring EGFRT790M mutation (IC50=1.32 µM) over A431 cells overexpressing EGFRWT (IC50=4.96 µM). EGFR-IN-60 exhibits good oral absorption, potent and safe antitumor activity. EGFR-IN-60 induces cell death through apoptosis supported by increased Bax/Bcl-2 ratio[1].

  • CAS Number: 2699877-43-3
  • MF: C28H28Cl2N6O
  • MW: 535.47
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

(R)​-​CR8

(R)​-​CR8 (CR8), a second-generation analog of Roscovitine, is a potent CDK1/2/5/7/9 inhibitor. (R)​-​CR8 (CR8) inhibits CDK1/cyclin B (IC50=0.09 μM), CDK2/cyclin A (0.072 μM), CDK2/cyclin E (0.041 μM), CDK5/p25 (0.11 μM), CDK7/cyclin H (1.1 μM), CDK9/cyclin T (0.18 μM) and CK1δ/ε (0.4 μM). (R)​-​CR8 (CR8) induces apoptosis and has neuroprotective effect[1][2].

  • CAS Number: 294646-77-8
  • MF: C24H29N7O
  • MW: 431.533
  • Catalog: Apoptosis
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 671.4±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 359.8±34.3 °C

HSP90-IN-13

HSP90-IN-13 (compound 5k) is a highly potent HSP90 pan inhibitor with an IC50 value of 25.07 nM. HSP90-IN-13 has multi-target activity against EGFR, VEGFR-2 and Topoisomerase-2. HSP90-IN-13 causes cell cycle arrest at G2/M phase and induces apoptosis of MCF-7 cells through mitochondrial-mediated pathway[1].

  • CAS Number: 2446055-29-2
  • MF: C26H21N5O3S
  • MW: 483.54
  • Catalog: Apoptosis
  • Density: N/A
  • Boiling Point: N/A
  • Melting Point: N/A
  • Flash Point: N/A

Cedrelone

Cedrelone, a limonoid, is a Phenazine biosynthesis-like domain-containing protein (PBLD) activator. Cedrelone induces cancer cell apoptosis, and possess antitumor effects[1].

  • CAS Number: 1254-85-9
  • MF: C26H30O5
  • MW: 422.51
  • Catalog: Apoptosis
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 584.2±50.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 307.1±30.1 °C

Oroxin B

Oroxin B (OB) is a flavonoid isolated from traditional Chinese herbal medicine Oroxylum indicum (L.) Vent. Oroxin B (OB) possesses obvious inhibitory effect and induces early apoptosis rather than late apoptosis on liver cancer cells through upregulation of PTEN, down regulation of COX-2, VEGF, PI3K, and p-AKT[1].Oroxin B (OB) selectively induces tumor-suppressive ER stress in malignant lymphoma cells[2].

  • CAS Number: 114482-86-9
  • MF: C27H30O15
  • MW: 594.518
  • Catalog: Apoptosis
  • Density: 1.8±0.1 g/cm3
  • Boiling Point: 957.5±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 318.1±27.8 °C

Tripchlorolide

Tripchlorolide is a neuroprotective agent that can be found in Tripterygium wilfordii. Tripchlorolide prevents tumor growth by inducing apoptosis and autophagy. Tripchlorolide improves cognitive deficits in Alzheimer's disease[1][2][3].

  • CAS Number: 132368-08-2
  • MF: C20H25ClO6
  • MW: 396.86
  • Catalog: Apoptosis
  • Density: 1.5±0.1 g/cm3
  • Boiling Point: 641.8±55.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 341.9±31.5 °C

Caspase-3/7 Inhibitor II

Ac-DNLD-CHO (Ac-Asp-Asn-Leu-Asp-CHO) is a Caspase-3/7 inhibitor (IC50: 9.89, 245 nM respectively; Kiapp: 0.68, 55.7 nM respectively). Ac-DNLD-CHO can be used for research of caspase-mediated apoptosis diseases, such as neurodegenerative disorders and viral infection diseases[1].

  • CAS Number: 775289-20-8
  • MF: C20H31N5O10
  • MW: 501.488
  • Catalog: Caspase
  • Density: 1.3±0.1 g/cm3
  • Boiling Point: 1034.0±65.0 °C at 760 mmHg
  • Melting Point: N/A
  • Flash Point: 579.1±34.3 °C