C.I. 74240
C.I. 74240 structure
|
Common Name | C.I. 74240 | ||
|---|---|---|---|---|
| CAS Number | 33864-99-2 | Molecular Weight | 722.32300 | |
| Density | N/A | Boiling Point | N/A | |
| Molecular Formula | C38H30CuN10S+ | Melting Point | 148ºC | |
| MSDS | N/A | Flash Point | N/A | |
|
Lrp4 domains differentially regulate limb/brain development and synaptic plasticity.
PLoS ONE 10(2) , e0116701, (2015) Apolipoprotein E (ApoE) genotype is the strongest predictor of Alzheimer's Disease (AD) risk. ApoE is a cholesterol transport protein that binds to members of the Low-Density Lipoprotein (LDL) Receptor family, which includes LDL Receptor Related Protein 4 (Lr... |
|
|
Filamin-interacting proteins, Cfm1 and Cfm2, are essential for the formation of cartilaginous skeletal elements.
Hum. Mol. Genet. 23(11) , 2953-67, (2014) Mutations of Filamin genes, which encode actin-binding proteins, cause a wide range of congenital developmental malformations in humans, mainly skeletal abnormalities. However, the molecular mechanisms underlying Filamin functions in skeletal system formation... |
|
|
The mych gene is required for neural crest survival during zebrafish development.
PLoS ONE 3(4) , e2029, (2008) Among Myc family genes, c-Myc is known to have a role in neural crest specification in Xenopus and in craniofacial development in the mouse. There is no information on the function of other Myc genes in neural crest development, or about any developmental rol... |
|
|
Genetic analysis of Hedgehog signaling in ventral body wall development and the onset of omphalocele formation.
PLoS ONE 6(1) , e16260, (2011) An omphalocele is one of the major ventral body wall malformations and is characterized by abnormally herniated viscera from the body trunk. It has been frequently found to be associated with other structural malformations, such as genitourinary malformations... |
|
|
Analysis and testing of biological stains--the Biological Stain Commission Procedures.
Biotech. Histochem. 77(5&6) , 237-275, (2002)
|
|
|
Lamin A-dependent misregulation of adult stem cells associated with accelerated ageing.
Nat. Cell Biol. 10(4) , 452-9, (2008) The premature-ageing disease Hutchinson-Gilford Progeria Syndrome (HGPS) is caused by constitutive production of progerin, a mutant form of the nuclear architectural protein lamin A. Progerin is also expressed sporadically in wild-type cells and has been link... |
|
|
Constitutive L-Sox5 overexpression delays differentiation of ATDC5 cells into chondrocytes and correlates with reduced expression of differentiation markers.
Mol. Cell Biochem. 401(1-2) , 21-6, (2015) L-Sox5 is a member of sex-determining region Y-type high mobility group box (SOX) family of transcription factors. We assessed the effects of retroviral overexpression of L-Sox5 on chondrocyte differentiation using the clonal murine cell line ATDC5. We observ... |
|
|
Discovery of candidate disease genes in ENU-induced mouse mutants by large-scale sequencing, including a splice-site mutation in nucleoredoxin.
PLoS Genet. 5(12) , e1000759, (2009) An accurate and precisely annotated genome assembly is a fundamental requirement for functional genomic analysis. Here, the complete DNA sequence and gene annotation of mouse Chromosome 11 was used to test the efficacy of large-scale sequencing for mutation i... |
|
|
Toxicological Characterization of the Inorganic and Organic Arsenic Metabolite Thio-DMA in Cultured Human Lung Cells.
J. Toxicol. 2011 , 373141, (2011) We synthesised and toxicologically characterised the arsenic metabolite thiodimethylarsinic acid (thio-DMA(V)). Successful synthesis of highly pure thio-DMA(V) was confirmed by state-of-the-art analytical techniques including (1)H-NMR, HPLC-FTMS, and HPLC-ICP... |
|
|
Notch pathway regulation of chondrocyte differentiation and proliferation during appendicular and axial skeleton development.
Proc. Natl. Acad. Sci. U. S. A. 106 , 14420-5, (2009) The role of Notch signaling in cartilage differentiation and maturation in vivo was examined. Conditional Notch pathway gain and loss of function was achieved using a Cre/loxP approach to manipulate Notch signaling in cartilage precursors and chondrocytes of ... |