3-(N-吗啉)丙磺酸
3-(N-吗啉)丙磺酸结构式
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常用名 | 3-(N-吗啉)丙磺酸 | 英文名 | MOPS |
|---|---|---|---|---|
| CAS号 | 1132-61-2 | 分子量 | 209.263 | |
| 密度 | 1.298 g/cm3 | 沸点 | N/A | |
| 分子式 | C7H15NO4S | 熔点 | 277-282 °C | |
| MSDS | 中文版 美版 | 闪点 | 116 °C | |
| 符号 |
GHS07 |
信号词 | Warning |
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Proteasome-mediated degradation of FRIGIDA modulates flowering time in Arabidopsis during vernalization.
Plant Cell 26(12) , 4763-81, (2015) Winter-annual accessions of Arabidopsis thaliana require either exposure to cold stress or vernalization to initiate flowering via FRIGIDA (FRI). FRI acts as a scaffold protein to recruit several chromatin modifiers that epigenetically modify flowering genes.... |
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The Mtr4 ratchet helix and arch domain both function to promote RNA unwinding.
Nucleic Acids Res. 42(22) , 13861-72, (2014) Mtr4 is a conserved Ski2-like RNA helicase and a subunit of the TRAMP complex that activates exosome-mediated 3'-5' turnover in nuclear RNA surveillance and processing pathways. Prominent features of the Mtr4 structure include a four-domain ring-like helicase... |
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Antisense oligonucleotides capable of promoting specific target mRNA reduction via competing RNase H1-dependent and independent mechanisms.
PLoS ONE 9(10) , e108625, (2014) Antisense oligonucleotides (ASOs) are most commonly designed to reduce targeted RNA via RNase H1-dependent degradation. In this paper we demonstrate that cellular proteins can compete for sites targeted by RNase H1-dependent ASOs. We further show that some AS... |
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Allelic effects on starch structure and properties of six starch biosynthetic genes in a rice recombinant inbred line population.
Rice (N. Y.) 8 , 15, (2015) The genetic diversity of six starch biosynthetic genes (Wx, SSI, SSIIa, SBEI, SBEIIa and SBEIIb) in indica and japonica rices opens an opportunity to produce a new variety with more favourable grain starch quality. However, there is limited information about ... |
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FGFR1 mediates recombinant thrombomodulin domain-induced angiogenesis.
Cardiovasc. Res. 105(1) , 107-17, (2014) The recombinant epidermal growth factor-like domain plus the serine/threonine-rich domain of thrombomodulin (rTMD23) promotes angiogenesis and accelerates the generation of activated protein C (APC), which facilitates angiogenesis. The aim of this study was t... |
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LRRK1-phosphorylated CLIP-170 regulates EGFR trafficking by recruiting p150Glued to microtubule plus ends.
J. Cell Sci. 128(2) , 385-96, (2015) The binding of ligand to epidermal growth factor receptor (EGFR) causes the receptor to become activated and stimulates the endocytosis of EGFR. Early endosomes containing activated EGFR migrate along microtubules as they mature into late endosomes. We have r... |
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Efficient programmable gene silencing by Cascade.
Nucleic Acids Res. 43(1) , 237-46, (2015) Methods that permit controlled changes in the expression of genes are important tools for biological and medical research, and for biotechnological applications. Conventional methods are directed at individually changing each gene, its regulatory elements or ... |
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DOCK8 regulates lymphocyte shape integrity for skin antiviral immunity.
J. Exp. Med. 211(13) , 2549-66, (2014) DOCK8 mutations result in an inherited combined immunodeficiency characterized by increased susceptibility to skin and other infections. We show that when DOCK8-deficient T and NK cells migrate through confined spaces, they develop cell shape and nuclear defo... |
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Molecular characterization of Plasmodium falciparum Bruno/CELF RNA binding proteins.
Mol. Biochem. Parasitol. 198(1) , 1-10, (2014) The human malaria parasite Plasmodium falciparum employs intricate post-transcriptional regulatory mechanisms in different stages of its life cycle. Despite the importance of post-transcriptional regulation, key elements of these processes, namely RNA binding... |
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Myocardial mitochondrial dysfunction in mice lacking adiponectin receptor 1.
Basic Res. Cardiol. 110 , 37, (2015) Hypoadiponectinemia is an independent predictor of cardiovascular disease, impairs mitochondrial function in skeletal muscle, and has been linked to the pathogenesis of Type 2 diabetes. In models of Type 2 diabetes, myocardial mitochondrial function is impair... |