乙酰基-缬氨酰-丙氨酰-天冬氨醛
乙酰基-缬氨酰-丙氨酰-天冬氨醛结构式
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常用名 | 乙酰基-缬氨酰-丙氨酰-天冬氨醛 | 英文名 | N-Acetyl-Val-Ala-Asp-al |
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| CAS号 | 147837-52-3 | 分子量 | 329.349 | |
| 密度 | 1.2±0.1 g/cm3 | 沸点 | 698.2±55.0 °C at 760 mmHg | |
| 分子式 | C14H23N3O6 | 熔点 | N/A | |
| MSDS | 中文版 美版 | 闪点 | 376.0±31.5 °C |
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zVAD-induced autophagic cell death requires c-Src-dependent ERK and JNK activation and reactive oxygen species generation.
Autophagy 7 , 217-228, (2011) The treatment of L929 fibrosarcoma cells with zVAD has been shown to induce necroptosis. However, whether autophagy is involved or not in this event remains controversial. In this study, we re-examined the role of autophagy in zVAD-induced cell death in L929 ... |
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Inhibition of human caspases by peptide-based and macromolecular inhibitors.
J. Biol. Chem. 273 , 32608-32613, (1998) Studies with peptide-based and macromolecular inhibitors of the caspase family of cysteine proteases have helped to define a central role for these enzymes in inflammation and mammalian apoptosis. A clear interpretation of these studies has been compromised b... |
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Caspase activation by BCR cross-linking in immature B cells: differential effects on growth arrest and apoptosis.
FASEB J. 13 , 931-944, (1999) The B cell lymphoma WEHI-231 has been used as a model to study immature B cell tolerance, based on its capacity to undergo growth arrest and programmed cell death on B cell receptor (BCR) cross-linking. Using this model to identify the molecular mechanisms un... |