乙烯基碳酸乙烯酯
乙烯基碳酸乙烯酯结构式
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常用名 | 乙烯基碳酸乙烯酯 | 英文名 | 4-Vinyl-1,3-dioxolan-2-one |
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| CAS号 | 4427-96-7 | 分子量 | 114.099 | |
| 密度 | 1.3±0.1 g/cm3 | 沸点 | 238.0±29.0 °C at 760 mmHg | |
| 分子式 | C5H6O3 | 熔点 | N/A | |
| MSDS | 中文版 美版 | 闪点 | 131.2±18.4 °C | |
| 符号 |
GHS06 |
信号词 | Danger |
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Foam cell-derived 4-hydroxynonenal induces endothelial cell senescence in a TXNIP-dependent manner.
J. Cell. Mol. Med. 19 , 1887-99, (2015) Vascular endothelial cell (VEC) senescence is considered an early event in the development of atherosclerotic lesions. Stressful stimuli, in particular oxidative stress, have been linked to premature senescence in the vasculature. Foam cells are a major sourc... |
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Regulation of valve endothelial cell vasculogenic network architectures with ROCK and Rac inhibitors.
Microvasc. Res. 98 , 108-18, (2015) The age- and disease-dependent presence of microvessels within heart valves is an understudied characteristic of these tissues. Neovascularization involves endothelial cell (EC) migration and cytoskeletal reorientation, which are heavily regulated by the Rho ... |
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3-Dimensional spatially organized PEG-based hydrogels for an aortic valve co-culture model.
Biomaterials 67 , 354-64, (2015) Physiologically relevant in vitro models are needed to study disease progression and to develop and screen potential therapeutic interventions for disease. Heart valve disease, in particular, has no early intervention or non-invasive treatment because there i... |
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Timing of galectin-1 exposure differentially modulates Nipah virus entry and syncytium formation in endothelial cells.
J. Virol. 89(5) , 2520-9, (2015) Nipah virus (NiV) is a deadly emerging enveloped paramyxovirus that primarily targets human endothelial cells. Endothelial cells express the innate immune effector galectin-1 that we have previously shown can bind to specific N-glycans on the NiV envelope fus... |
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Laminin Peptide-Immobilized Hydrogels Modulate Valve Endothelial Cell Hemostatic Regulation.
PLoS ONE 10 , e0130749, (2015) Valve endothelial cells (VEC) have unique phenotypic responses relative to other types of vascular endothelial cells and have highly sensitive hemostatic functions affected by changes in valve tissues. Furthermore, effects of environmental factors on VEC hemo... |
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Venous Endothelial Marker COUP-TFII Regulates the Distinct Pathologic Potentials of Adult Arteries and Veins.
Sci. Rep. 5 , 16193, (2015) Arteries and veins have very different susceptibility to certain vascular diseases such as atherosclerosis and vascular calcification. The molecular mechanisms of these differences are not fully understood. In this study, we discovered that COUP-TFII, a trans... |
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Valvular interstitial cells suppress calcification of valvular endothelial cells.
Atherosclerosis 242 , 251-60, (2015) Calcific aortic valve disease (CAVD) is the most common heart valve disease in the Western world. We previously proposed that valvular endothelial cells (VECs) replenish injured adult valve leaflets via endothelial-to-mesenchymal transformation (EndMT); howev... |
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Magnetic ferroferric oxide nanoparticles induce vascular endothelial cell dysfunction and inflammation by disturbing autophagy.
J. Hazard. Mater. 304 , 186-95, (2015) Despite the considerable use of magnetic ferroferric oxide nanoparticles (Fe3O4NPs) worldwide, their safety is still an important topic of debate. In the present study, we detected the toxicity and biological behavior of bare-Fe3O4NPs (B-Fe3O4NPs) on human um... |
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Endothelial cells obtained from patients affected by chronic venous disease exhibit a pro-inflammatory phenotype.
PLoS ONE 7 , e39543, (2012) The inflammatory properties of vein endothelium in relation to chronic venous disease (CVD) have been poorly investigated. Therefore, new insights on the characteristics of large vein endothelium would increase our knowledge of large vessel physiopathology.Su... |
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Angiogenic sprouting requires the fine tuning of endothelial cell cohesion by the Raf-1/Rok-α complex.
Dev. Cell 22 , 158-71, (2012) Sprouting angiogenesis, crucial for the development of new blood vessels, is a prime example of collective migration in which endothelial cells migrate as a group joined via cadherin-containing adherens junctions (AJ). The actomyosin apparatus is connected to... |