SLIGKV-NH2
SLIGKV-NH2 structure
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Common Name | SLIGKV-NH2 | ||
|---|---|---|---|---|
| CAS Number | 190383-13-2 | Molecular Weight | 614.77800 | |
| Density | N/A | Boiling Point | N/A | |
| Molecular Formula | C28H54N8O7 | Melting Point | N/A | |
| MSDS | Chinese USA | Flash Point | N/A | |
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The effect of kinin B1 receptor on chronic itching sensitization.
Mol. Pain 11 , 70, (2015) Altered kallikrein-related peptidase activity and bradykinin are associated with skin disorders in humans and mice under chronic inflammation conditions. The bradykinin B1 receptor (B1R), also known as one of the G-protein-coupled receptor family and usually ... |
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Heparin inhibits melanosome uptake and inflammatory response coupled with phagocytosis through blocking PI3k/Akt and MEK/ERK signaling pathways in human epidermal keratinocytes.
Pigment Cell Melanoma Res. 27(6) , 1063-74, (2014) To gain insight for the role of mast cell-produced heparin in the regulation of epidermal homeostasis and skin pigmentation, we have investigated the effect of heparin on melanosome uptake and proinflammatory responses in normal human epidermal keratinocytes ... |
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Activation of TRPV1 mediates thymic stromal lymphopoietin release via the Ca2+/NFAT pathway in airway epithelial cells.
FEBS Lett. 588(17) , 3047-54, (2014) The airway epithelium is exposed to a range of irritants in the environment that are known to trigger inflammatory response such as asthma. Transient receptor potential vanilloid 1 (TRPV1) is a Ca(2+)-permeable cation channel critical for detecting noxious st... |
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Intestinal protease-activated receptor-2 and fecal serine protease activity are increased in canine inflammatory bowel disease and may contribute to intestinal cytokine expression.
J. Vet. Med. Sci. 76(8) , 1119-27, (2014) Serine proteases elicit cellular responses via protease-activated receptor-2 (PAR-2) which is known to regulate inflammation and the immune response. Although the gastrointestinal tract is exposed to large amounts of proteolytic enzymes, the role of PAR-2 in ... |
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Thrombin Augments LPS-Induced Human Endometrial Endothelial Cell Inflammation via PAR1 Activation.
Am. J. Reprod. Immunol. 76 , 29-37, (2016) Risk factors for preterm birth include placental abruption, giving rise to excessive decidual thrombin, and intrauterine bacterial infection. Human endometrial endothelial cells (HEECs) express Toll-like receptors (TLRs), and infection-derived agonists trigge... |