脱硫硫酸肝素(肝素IH)钠盐
脱硫硫酸肝素(肝素IH)钠盐结构式
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常用名 | 脱硫硫酸肝素(肝素IH)钠盐 | 英文名 | DE-N-SULFATED HEPARIN SODIUM SALT |
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| CAS号 | 61932-66-9 | 分子量 | 366.144 | |
| 密度 | N/A | 沸点 | N/A | |
| 分子式 | C10H13BrFN5O4 | 熔点 | N/A | |
| MSDS | 美版 | 闪点 | N/A |
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Complement activation in the brain after experimental intracerebral hemorrhage.
J. Neurosurg. 92 , 1016-1022, (2000) Brain edema formation following intracerebral hemorrhage (ICH) appears to be partly related to erythrocyte lysis and hemoglobin release. Erythrocyte lysis may be mediated by the complement cascade, which then triggers parenchymal injury. In this study the aut... |
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Nonanticoagulant heparin prevents coronary endothelial dysfunction after brief ischemia-reperfusion injury in the dog.
Circulation 99 , 1062-1068, (1999) Coronary endothelial dysfunction after brief ischemia-reperfusion (IR) remains a clinical problem. We investigated the role of heparin and N-acetylheparin, a nonanticoagulant heparin derivative, in modulating coronary endothelial function after IR injury, wit... |
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Non-anticoagulant heparin increases endothelial nitric oxide synthase activity: role of inhibitory guanine nucleotide proteins.
J. Mol. Cell. Cardiol. 30 , 2669-2682, (1998) Heparin, which is widely used clinically, has recently been shown to have specific properties affecting the vascular endothelium. We hypothesized that heparin stimulates endothelial nitric oxide synthase (eNOS) activity by a mechanism independent of its antic... |
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N-Acetylheparin pretreatment reduces infarct size in the rabbit.
Pharmacology 58 , 120-131, (1999) The ability of the heparin derivative, N-acetylheparin (NHEP) to protect the heart from regional ischemia/reperfusion injury was examined in vivo. NHEP (2 mg/kg i.v.) or vehicle was administered 2 h before occlusion of the left circumflex coronary (LCX) arter... |