Influenza Hemagglutinin (HA) Peptide
Influenza Hemagglutinin (HA) Peptide structure
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Common Name | Influenza Hemagglutinin (HA) Peptide | ||
|---|---|---|---|---|
| CAS Number | 92000-76-5 | Molecular Weight | 1102.15000 | |
| Density | 1.414±0.06 g/cm3 (20ºC 760 Torr) | Boiling Point | N/A | |
| Molecular Formula | C53H67N9O17 | Melting Point | N/A | |
| MSDS | Chinese USA | Flash Point | N/A | |
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Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation.
Nat. Commun. 5 , 4664, (2014) The mechanisms underlying cardiac automaticity are still incompletely understood and controversial. Here we report the complete conditional and time-controlled silencing of the 'funny' current (If) by expression of a dominant-negative, non-conductive HCN4-cha... |
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SUMOylation of Syntaxin1A regulates presynaptic endocytosis.
Sci. Rep. 5 , 17669, (2015) Neurotransmitter release from the presynaptic terminal is under very precise spatial and temporal control. Following neurotransmitter release, synaptic vesicles are recycled by endocytosis and refilled with neurotransmitter. During the exocytosis event leadin... |
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Exosome derived from murine adipose-derived stromal cells: Neuroprotective effect on in vitro model of amyotrophic lateral sclerosis.
Exp. Cell Res. 340 , 150-8, (2016) Therapeutic strategies for the fatal neurodegenerative disease amyotrophic lateral sclerosis (ALS) have not yet provided satisfactory results. Interest in stem cells for the treatment of neurodegenerative diseases is increasing and their beneficial action see... |
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HIV-1 Vpr redirects host ubiquitination pathway.
J. Virol. 88(16) , 9141-52, (2014) HIV-1 modulates key host cellular pathways for successful replication and pathogenesis through viral proteins. By evaluating the hijacking of the host ubiquitination pathway by HIV-1 at the whole-cell level, we now show major perturbations in the ubiquitinate... |
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Distinct activation of an E2 ubiquitin-conjugating enzyme by its cognate E3 ligases.
Proc. Natl. Acad. Sci. U. S. A. 112(7) , E625-32, (2015) A significant portion of ubiquitin (Ub)-dependent cellular protein quality control takes place at the endoplasmic reticulum (ER) in a process termed "ER-associated degradation" (ERAD). Yeast ERAD employs two integral ER membrane E3 Ub ligases: Hrd1 (also term... |
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Emergence of an Ancestral Glycoprotein Hormone in the Pituitary of the Sea Lamprey, a Basal Vertebrate.
Endocrinology 156 , 3026-37, (2015) The gnathostome (jawed vertebrates) classical pituitary glycoprotein hormones, FSH, LH, and TSH, consist of a common α-subunit (GpA1) and unique β-subunits (Gpβ1, -2, and -3), whereas a recently identified pituitary glycoprotein hormone, thyrostimulin, consis... |
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Intravenous administration of brain-targeted stable nucleic acid lipid particles alleviates Machado-Joseph disease neurological phenotype.
Biomaterials 82 , 124-37, (2016) Others and we showed that RNA interference holds great promise for the treatment of dominantly inherited neurodegenerative disorders such as Machado-Joseph disease (MJD), for which there is no available treatment. However, successful experiments involved intr... |
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Expression, regulation and functional assessment of the 80 amino acid Small Adipocyte Factor 1 (Smaf1) protein in adipocytes.
Arch. Biochem. Biophys. 590 , 27-36, (2016) The gene for Small Adipocyte Factor 1, Smaf1 (also known as adipogenin, ADIG), encodes a ∼600 base transcript that is highly upregulated during 3T3-L1 in vitro adipogenesis and markedly enriched in adipose tissues. Based on the lack of an obvious open reading... |
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Impaired adipogenic capacity in induced pluripotent stem cells from lipodystrophic patients with BSCL2 mutations.
Metab. Clin. Exp. 65 , 543-56, (2016) Congenital generalized lipodystrophy (CGL) is an autosomal recessive disorder characterized by marked scarcity of adipose tissue, extreme insulin resistance, hypertriglyceridemia, hepatic steatosis and early-onset diabetes. Mutation of the BSCL2/SEIPIN gene c... |
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Staufen1 Regulates Multiple Alternative Splicing Events either Positively or Negatively in DM1 Indicating Its Role as a Disease Modifier.
PLoS Genet. 12 , e1005827, (2016) Myotonic dystrophy type 1 (DM1) is a neuromuscular disorder caused by an expansion of CUG repeats in the 3' UTR of the DMPK gene. The CUG repeats form aggregates of mutant mRNA, which cause misregulation and/or sequestration of RNA-binding proteins, causing a... |