NPAS3-IN-1
Modify Date: 2025-08-27 07:41:00
NPAS3-IN-1 structure
|
Common Name | NPAS3-IN-1 | ||
|---|---|---|---|---|
| CAS Number | 2207-44-5 | Molecular Weight | 295.36100 | |
| Density | N/A | Boiling Point | N/A | |
| Molecular Formula | C10H5N3O2S3 | Melting Point | N/A | |
| MSDS | Chinese USA | Flash Point | N/A | |
| Symbol |
GHS06 |
Signal Word | Danger | |
Use of NPAS3-IN-1NPAS3-IN-1 is a potent inhibitor of NPAS3-ARNT heterodimerization and regulates NPAS3 transcription by disrupting the heterodimerization of NPAS3 with ARNT at the cellular level[1]. |
| Name | 2-(5-nitrothiazol-2-ylthio)benzo[d]thiazole |
|---|---|
| Synonym | More Synonyms |
| Description | NPAS3-IN-1 is a potent inhibitor of NPAS3-ARNT heterodimerization and regulates NPAS3 transcription by disrupting the heterodimerization of NPAS3 with ARNT at the cellular level[1]. |
|---|---|
| Related Catalog | |
| References |
| Molecular Formula | C10H5N3O2S3 |
|---|---|
| Molecular Weight | 295.36100 |
| Exact Mass | 294.95400 |
| PSA | 153.38000 |
| LogP | 4.33540 |
| Symbol |
GHS06 |
|---|---|
| Signal Word | Danger |
| Hazard Statements | H301-H319 |
| Precautionary Statements | P301 + P310-P305 + P351 + P338 |
| RIDADR | UN 2811 6.1 / PGIII |
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DUSP4 deficiency caused by promoter hypermethylation drives JNK signaling and tumor cell survival in diffuse large B cell lymphoma.
J. Exp. Med. 212 , 775-92, (2015) The epigenetic dysregulation of tumor suppressor genes is an important driver of human carcinogenesis. We have combined genome-wide DNA methylation analyses and gene expression profiling after pharmac... |
| BI-87G3 |
| 2-(5-nitro-thiazol-2-ylsulfanyl)-benzothiazole |
| 5'-Nitro-2'-thiazolyl-2-benzothiazolylsulfiol |
| JNK INHIBITOR XI |