促甲状腺素结构式
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常用名 | 促甲状腺素 | 英文名 | tsh |
|---|---|---|---|---|
| CAS号 | 9002-71-5 | 分子量 | 422.436 | |
| 密度 | N/A | 沸点 | N/A | |
| 分子式 | C18H26N6O6 | 熔点 | N/A | |
| MSDS | 美版 | 闪点 | N/A |
促甲状腺素用途促甲状腺激素(TSH)是由垂体前叶的促甲状腺细胞产生的一种促甲状腺激素。促甲状腺素调节甲状腺的内分泌功能[1][2]。 |
| 中文名 | 促甲状腺素 |
|---|---|
| 英文名 | Thyrotropic hormone from bovine pituitary |
| 英文别名 | 更多 |
| 描述 | 促甲状腺激素(TSH)是由垂体前叶的促甲状腺细胞产生的一种促甲状腺激素。促甲状腺素调节甲状腺的内分泌功能[1][2]。 |
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| 相关类别 | |
| 参考文献 |
| 分子式 | C18H26N6O6 |
|---|---|
| 分子量 | 422.436 |
| 精确质量 | 422.191376 |
| 外观性状 | lyophilized powder |
| 储存条件 | 密封保存 |
| 稳定性 | 1.遇蛋白水解酶、胱氨酸、氧化剂 ( 如高锰酸钾、碘)等失活。 2.TSH过多时甲状腺增生,血管丰富。TSH的作用是它活化甲状腺内的腺苷酸环化酶,生成cAMP作为第二信使而产生的。 |
| 更多 | 1.性状:白色或淡黄色粉末。 2.溶解性:溶于水。 |
| 危害码 (欧洲) | B |
|---|---|
| 危险品运输编码 | NONH for all modes of transport |
| WGK德国 | 3 |
| RTECS号 | XP3581600 |
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Excess iodide induces an acute inhibition of the sodium/iodide symporter in thyroid male rat cells by increasing reactive oxygen species.
Endocrinology 156(4) , 1540-51, (2015) Na+/I- symporter (NIS) mediates iodide (I-) uptake in the thyroid gland, the first and rate-limiting step in the biosynthesis of the thyroid hormones. The expression and function of NIS in thyroid cel... |
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Effects of magnesium on cytomorphology and enzyme activities in thyroid of rats.
Indian J. Exp. Biol. 52(8) , 787-92, (2014) Till date knowledge regarding the effects of high dietary magnesium on thyroid gland is incomprehensive though certain epidemiological studies reported development of thyroid gland dysfunctions in peo... |
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Extreme phenotypic variability of thyroid dysgenesis in six new cases of congenital hypothyroidism due to PAX8 gene loss-of-function mutations.
Eur. J. Endocrinol. 171(4) , 499-507, (2014) Within the last two decades, heterozygous loss-of-function PAX8 mutations have been reported in patients with a wide degree of thyroid gland dysfunction and growth despite the presence of identical mu... |
| L-Prolinamide, 5-oxo-L-prolyl-L-histidyl-, acetate (1:1) |
| MFCD00166060 |
| 5-Oxo-L-prolyl-L-histidyl-L-prolinamide acetate (1:1) |
| EINECS 232-664-4 |