A series of phenyl sulfone substituted quinoxaline were prepared and the lead compound 13 (WYE-672) was shown to be a tissue selective LXR Agonist. Compound 13 demonstrated partial agonism for LXRβ in kidney HEK-293 cells but did not activate Gal4 LXRβ fusion proteins in huh-7 liver cells. Although 13 showed potent binding affinity to LXRβ (IC50= 53 nM), it had little binding affinity for LXRα (IC50> 1.0 μM) and did not recruit any coactivator/ ...